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ACETYL L-CARNITINE 200g by GEN-TEC

$59.99

$44.99

Stock: Available

Product Code: GENTALC

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ACETYL L-CARNITINE

100% ACETYL L-CARNITINE POWDER
BASIC FUNCTIONS Fat metabolism, cognitive functions (ie reflex’s and co-ordination), vigilance, fatloss, mind/muscle connection.

Acetyl L-Carnitine (ALC) plays a critical role in not only lipid and energy metabolism but also within nerve cells of the central nervous system (CNS) (Rebouche, 2012). Research shows that in the periphery skeletal muscle sites,  ALC is a co-factor for beta-oxidation (metabolism of fatty acids)whereby it mops up available fats to be sent to the muscle cells for metabolism (Stephens et al., 2007, Kido et al., 2001). More specifically it translocates long chain fatty acids (LCFA) to the mitochondrial matrix where it can undergo beta –oxidation and enter the Kreb cycle for fat derived ATP re-synthesis (Stephens et al., 2007). This involvement in fat metabolism is also one way by which ALC assists with energy levels during exercise.

It is important to understand that the mitochondria is an organelle within a cell that functions to produce energy by ATP synthesis from primarily fats and carbohydrates (Stephens et al., 2007). If the mitochondria is a factory to burn fuel then ALC is the employee who transports that fuel into the factory to be burned. If there is insufficient ALC (employees) then naturally there is a reduced mitochondrial  (factory) use of fats (fuel). In addition,  ALC can reduce the amount of muscle damage experienced by intense exercise by acting as “mitochondrial antioxidant”, which if revisiting the previous factory analogy, within every factory there must be some level of maintenance  to ensure the longevity and proper functioning of machinery, so ALC is like that ‘oil’ or ‘maintenance check’ to ensure a healthy and functioning environment for fats (fuel) to be burned (Stephens et al., 2007).

Moreover, ALC crosses the blood brain barrier which means it exhibits activity in not only the muscle cells for energy but also in the brain and nerve cells (Kido et al., 2001).  Over recent years research has revealed that ALC acts as a neuro-protective agent against alcohol related nerve cell damage and glucose transportation to the brain (Abdul Muneer et al., 2011). Furthermore, evidence has indicated possible clinical benefit  for ALC use in neurodegenerative conditions such as Alzheimer’s disease, diabetic neuropathy and other conditions resulting in nerve damage (Malaguarnera, 2012, Ruggenenti et al., 2009). It appears that ALC assists with the activity of cholinergic neurons, membrane stabilization and mitochondrial function (Palacios et al., 2011).  To further understand how, we must understand that the brain and nerve cells also require adequate energy supply, so they too poses mitochondria within the nerve cells (Palacios et al., 2011).


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